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• W2014107202 abstract "We earlier proposed that a human endogenous retroviral (HERV) superantigen (SAg) IDDMK1,222 may cause type I diabetes by activating autoreactive T cells. Viral infections and induction of interferon-α (IFN-α) are tightly associated with the onset of autoimmunity. Here we establish a link between viral infections and IFN-α-regulated SAg expression of the polymorphic and defective HERV-K18 provirus. HERV-K18 has three alleles, IDDMK1,222 and two full-length envelope genes, that all encode SAgs. Expression of HERV-K18 SAgs is inducible by IFN-α and this is sufficient to stimulate Vβ7 T cells to levels comparable to transfectants constitutively expressing HERV-K18 SAgs. Endogenous SAgs induced via IFN-α by viral infections is a novel mechanism through which environmental factors may cause disease in genetically susceptible individuals." @default.
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• W2014107202 date "2001-10-01" @default.
• W2014107202 modified "2023-10-16" @default.
• W2014107202 title "Interferon-α-Induced Endogenous Superantigen" @default.
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• W2014107202 doi "https://doi.org/10.1016/s1074-7613(01)00212-6" @default.
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