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• W2014230992 endingPage "1935" @default.
• W2014230992 startingPage "1928" @default.
• W2014230992 abstract "The effects of a pomegranate byproduct (PBP, which includes the whole pomegranate fruit left after juice preparation) on atherosclerosis development in apolipoprotein E-deficient (E°) mice were studied. Consumption of PBP (17 or 51.5 μg of gallic acid equiv/kg/day) by the mice resulted in a significant reduction in atherosclerotic lesion size by up to 57%. PBP consumption significantly reduced oxidative stress in the mice peritoneal macrophages (MPM): Cellular lipid peroxide content decreased by up to 42%, the reduced glutathione levels increased by up to 53%, and paraoxonase 2 lactonase activity increased by up to 50%, as compared to MPM from E° mice that consumed only water. Furthermore, oxidized low-density lipoprotein (Ox-LDL) uptake by the MPM was reduced by up to 19%. Similar results were observed also in vitro. Treatment of J774A.1 macrophages with PBP (10 or 50 μmol/L of total polyphenols) significantly decreased both cellular total peroxide content and Ox-LDL uptake. It was thus concluded that PBP significantly attenuates atherosclerosis development by its antioxidant properties. Keywords: Pomegranate; macrophages; oxidative stress; apolipoprotein E-deficient mice; atherosclerosis" @default.
• W2014230992 created "2016-06-24" @default.
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• W2014230992 date "2006-02-02" @default.
• W2014230992 modified "2023-09-23" @default.
• W2014230992 title "Pomegranate Byproduct Administration to Apolipoprotein E-Deficient Mice Attenuates Atherosclerosis Development as a Result of Decreased Macrophage Oxidative Stress and Reduced Cellular Uptake of Oxidized Low-Density Lipoprotein" @default.
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• W2014230992 doi "https://doi.org/10.1021/jf0528207" @default.
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