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- W2014282354 endingPage "354" @default.
- W2014282354 startingPage "347" @default.
- W2014282354 abstract "Rheumatoid arthritis (RA) is a chronic debilitating disease of the joints. Both the innate and adaptive immune responses participate in the development and progression of RA. While several therapeutic reagents, such as TNF-α agonists, have been successfully developed for the clinical use in the treatment of RA, more than half of the patients do not respond to anti-TNF therapy. Therefore, new therapeutic reagents are needed. Recent studies have shown that sirtuin 1 (Sirt1), a nicotinamide adenine dinucleotide (NAD)-dependent histone deacetylase, is a critical negative regulator of both the innate and adaptive immune response in mice, and its altered functions are likely to be involved in autoimmune diseases. Small molecules that modulate Sirt1 functions are potential therapeutic reagents for autoimmune inflammatory diseases. This review highlights the role of Sirt1 in immune regulation and RA." @default.
- W2014282354 created "2016-06-24" @default.
- W2014282354 creator A5043224088 @default.
- W2014282354 creator A5057143739 @default.
- W2014282354 creator A5057577859 @default.
- W2014282354 date "2013-07-01" @default.
- W2014282354 modified "2023-10-16" @default.
- W2014282354 title "The Class III Histone Deacetylase Sirtuin 1 in Immune Suppression and Its Therapeutic Potential in Rheumatoid Arthritis" @default.
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