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- W2014398182 abstract "The proto-oncogene c-myc is a central regulator of cellular proliferation and cell growth, which executes its multiple activities mostly through transcriptional regulation of its target genes. Although the role of c-myc in the activation of transcription is clearly established, the mechanisms of transcriptional repression by c-myc are not well understood. It has been proposed that c-myc represses gene transcription by at least two distinct mechanisms (Gartel and Shchors, 2003). One mechanism is limited to the binding of c-myc to the DNA-binding protein Miz-1, via its C-terminal domain, resulting in inhibition of Miz-1 transcriptional activity (Staller et al., 2001). The other mechanism is dependent on c-myc binding to the Sp1 transcription factor through its central region leading to inhibition of Sp1 transcriptional activity (Gartel et al., 2001). Interestingly, both Sp1 and Miz-1 bind to the proximal promoter of the p21 gene (Gartel et al., 2001; Seoane et al., 2002), which is a major target of Myc repression (Coller et al., 2000)." @default.
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- W2014398182 date "2005-09-19" @default.
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- W2014398182 title "A new mode of transcriptional repression by c-myc: methylation" @default.
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- W2014398182 doi "https://doi.org/10.1038/sj.onc.1209101" @default.
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