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• W2014432319 endingPage "89" @default.
• W2014432319 startingPage "83" @default.
• W2014432319 abstract "Listeria monocytogenes is a facultative intracellular pathogen which is internalized by host mammalian cells upon binding to their surface. Further listerial growth occurs in the cytosol after escape from the phagosomal-endosomal compartment. We have previously reported that C1q is able to potentiate L. monocytogenes phagocytosis upon bacterial opsonization by ingestion through C1q-binding structures. In this report, we analysed the post-phagocytic events upon internalization of C1q-opsonized L. monocytogenes and found an induction of macrophage (Mphi)-like IC-21 cell bactericidal mechanisms displayed by the production of oxygen and nitrogen metabolites. Both types of molecules are effective in L. monocytogenes killing. Further analysis of the cellular responses promoted by interaction of C1q with its surface binding structures, leads us to consider C1q as a collaborative molecule involved in Mphi activation. Upon interaction with surface binding structures, C1q was able to trigger and/or amplify the production of reactive oxygen and nitrogen intermediates induced by stimuli such as interferon-gamma and L. monocytogenes phagocytosis." @default.
• W2014432319 created "2016-06-24" @default.
• W2014432319 creator A5012219203 @default.
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• W2014432319 creator A5029456446 @default.
• W2014432319 creator A5084927731 @default.
• W2014432319 date "2000-09-01" @default.
• W2014432319 modified "2023-10-17" @default.
• W2014432319 title "The contribution of both oxygen and nitrogen intermediates to the intracellular killing mechanisms of C1q-opsonized<i>Listeria monocytogenes</i>by the macrophage-like IC-21 cell line" @default.
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• W2014432319 doi "https://doi.org/10.1046/j.1365-2567.2000.00083.x" @default.
• W2014432319 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2327058" @default.
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