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• W2014700275 endingPage "39" @default.
• W2014700275 startingPage "39" @default.
• W2014700275 abstract "New insights from animal models of colon cancer: inflammation control as a new facet on the tumor suppressor APC gem Maged Zeineldin, Kristi L Neufeld Department of Molecular Biosciences, University of Kansas, Lawrence, KS, USA Abstract: Colorectal cancer (CRC) is one of the most common causes of cancer-related deaths worldwide. As with other cancers, CRC is a genetic disease, however, several risk factors including diet and chronic colitis predispose to the disease. Mutations in the tumor suppressor adenomatous polyposis coli (APC) initiate most cases of CRC. Recent data from mouse models suggest that APC mutations and colitis are not completely independent factors in colorectal carcinogenesis. Here, we review the evidence supporting an interaction between APC mutations and chronic colitis. We will also discuss possible pathophysiologic mechanisms behind this interaction. Keywords: rodent model, colon cancer, adenomatous polyposis coli, APC, tumor suppressor, inflammatory bowel disease&nbsp" @default.
• W2014700275 created "2016-06-24" @default.
• W2014700275 creator A5034601651 @default.
• W2014700275 creator A5037119065 @default.
• W2014700275 date "2015-01-01" @default.
• W2014700275 modified "2023-09-24" @default.
• W2014700275 title "New insights from animal models of colon cancer: inflammation control as a new facet on the tumor suppressor APC gem" @default.
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