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• W2014717772 abstract "1 This study was carried out in order to identify the receptor responsible for adenosine-induced dilatation of the hepatic arterial vascular bed. 2 Livers of 10 New Zealand White rabbits were perfused in vitro with Krebs-Bülbring buffer via the hepatic artery and the portal vein at constant flows of 26 and 77 ml min−1 100 g−1 liver respectively. The tone of the preparation was raised by the presence of noradrenaline in the perfusate (concentration: 10−5 m). 3 Dose-response curves for adenosine and its analogues 5′-N-ethyl-carboxamido-adenosine (NECA), the 2-substituted NECA analogue CGS 21680C, and R- and S-N6-phenyl-isopropyl-adenosine (R- and S-PIA) were obtained after their injection into the hepatic arterial supply. 4 The order of vasodilator potency of these agents was: NECA > CGS 21680C > adenosine > R-PIA > S-PIA. Their potency, expressed relative to that of adenosine, was in the approximate ratio 10:3:1:0.3:0.1, consistent with that resulting from activation of P1-purinoceptors of the A2 sub-type (which mediate vasodilatation due to adenosine). 5 The P1-purinoceptor antagonist 8-phenyltheophylline (10−5 m) caused significant attenuation of the vasodilatation to adenosine and analogues. 6 It is concluded that adenosine-induced dilatation of the hepatic arterial vascular bed is mediated by P1-purinoceptors of the A2 sub-type." @default.
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• W2014717772 date "1991-05-01" @default.
• W2014717772 modified "2023-09-23" @default.
• W2014717772 title "Adenosine-induced dilatation of the rabbit hepatic arterial bed is mediated by A2-purinoceptors" @default.
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• W2014717772 doi "https://doi.org/10.1111/j.1476-5381.1991.tb12307.x" @default.
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