Matches in SemOpenAlex for { <https://semopenalex.org/work/W2014862688> ?p ?o ?g. }
- W2014862688 abstract "Progression of diabetic nephropathy (DN) is manifested by gradual scarring of both the renal glomerulus and tubulointerstitial region. Over the past several years, the general understanding of the pathogenic factors that lead to renal fibrosis in DN has expanded considerably. In this review, some of the important factors that appear to be involved in driving this fibrosing process are discussed, with special emphasis on newer findings and insights. It is now clear that multiple cell types in the kidney contribute to progressive fibrosis in DN. New concepts about bradykinin, TGF-beta and eNOS signaling as well as JAK/STAT activation and the central role of inflammation in both glomerular and tubulointerstitial fibrosis are discussed." @default.
- W2014862688 created "2016-06-24" @default.
- W2014862688 creator A5003676386 @default.
- W2014862688 date "2008-08-23" @default.
- W2014862688 modified "2023-10-16" @default.
- W2014862688 title "New insights into the mechanisms of fibrosis and sclerosis in diabetic nephropathy" @default.
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- W2014862688 doi "https://doi.org/10.1007/s11154-008-9100-6" @default.
- W2014862688 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3776415" @default.
- W2014862688 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18726161" @default.
- W2014862688 hasPublicationYear "2008" @default.
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