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• W2014862976 abstract "Abstract This paper addresses the capacity of naive, effector, and memory CD4 T cells to control growth of a major histocompatibility complex (MHC) class II—positive B-cell lymphoma in vivo. To assess the role of T cells on their own without contributions by B cells, antibodies, or natural killer (NK) cells, we generated pure effector or memory CD4 T cells in Rag–/–gc–/– mice deficient in endogenous lymphocytes and NK cells. Lymphoma cells expressing a model antigen were injected into mice with T cells of cognate specificity that were either naive or in effector or resting memory state. Naive T cells were unable to prevent tumor growth, probably due to delay of efficient cross-presentation by dendritic cells. However, both effector and memory T cells, dependent on the amount of antigen available, controlled the tumor for a considerable period of time without the need for dendritic cell stimulation. Nevertheless, the tumor eventually grew uncontrolled in all cases. This was not because of a defect in T-cell homing to the tumor site or loss of MHC class II or costimulatory molecules by the tumor, but reflected mutual paralysis of T-cell responsiveness and antigen processing by tumor cells." @default.
• W2014862976 created "2016-06-24" @default.
• W2014862976 creator A5059045389 @default.
• W2014862976 creator A5086005310 @default.
• W2014862976 date "2003-06-01" @default.
• W2014862976 modified "2023-09-30" @default.
• W2014862976 title "Tumor and CD4 T-cell interactions: tumor escape as result of reciprocal inactivation" @default.
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• W2014862976 doi "https://doi.org/10.1182/blood-2002-10-3030" @default.
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