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- W2014984654 endingPage "136" @default.
- W2014984654 startingPage "125" @default.
- W2014984654 abstract "The role of chemokines in mediating directional cell migration is well established, but more recently it has become evident that chemokines are able to couple to distinct signalling pathways that are involved in not only chemotaxis, but also cell growth and transcriptional activation. The signalling pathway controlled by the phosphoinositide 3-kinase (PI3K) family of lipid kinases has been the focus of much attention with respect to their role in chemokine-mediated functional responses. Indeed, there now exists convincing biochemical, pharmacological and genetic evidence that both CC and CXC chemokines stimulate PI3K-dependent chemotaxis of inflammatory cells such as eosinophils, macrophages, neutrophils and T lymphocytes. This review considers the role of individual PI3Ks (e.g. the p85/p110 heterodimer, PI3Kgamma and PI3KC2alpha) as well their downstream effector targets in mediating chemokine-stimulated cell migration." @default.
- W2014984654 created "2016-06-24" @default.
- W2014984654 creator A5009500424 @default.
- W2014984654 creator A5025685997 @default.
- W2014984654 creator A5029868837 @default.
- W2014984654 date "2002-02-01" @default.
- W2014984654 modified "2023-10-18" @default.
- W2014984654 title "Chemokine signalling: pivoting around multiple phosphoinositide 3-kinases" @default.
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