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- W2015153989 abstract "Background: We have reported that TNF-α caused down-regulation of transient outward current (Ito) as well as hypertrophy in cultured neonatal myocytes. Although TNF-α induced hypertrophy was reported to be mediated by the activation of reactive oxygen speicies (ROS), the mechanism regulating ion channel is unclear. In this study, we evaluated the effect of n-acetyl cystein (NAC), an antioxidant, on TNF-α induced reduction of Ito and Ito-related molecules, and the regulation of action potential duration (APD) in neonatal rat myocytes." @default.
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- W2015153989 date "2005-05-01" @default.
- W2015153989 modified "2023-10-16" @default.
- W2015153989 title "TNF-α induces ion channel (Ito) down-regulation in myocyte independently on the Reactive Oxygen Species (ROS) generation" @default.
- W2015153989 doi "https://doi.org/10.1016/j.hrthm.2005.02.799" @default.
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