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- W2015223821 abstract "AbstractIn the last decade or so, an impairment of Hypolhalamic-Pituitary-Adrenal (HPA) axis activity has been observed in fatigue syndromes. Elevated levels of glucocorticoids help to prevent the immune system from over-reacting and generating a damaging autoimmune process. The corollary should be that reduced activity of the HPA axis and diminished levels of plasma corlisol could be associated with autoimmune (AI) disease. Experimental work in mice and rats supports this view. Furthermore, plasma levels of cortisol have been found to be low in the early stages of rheumatoid arthritis. There is some clinical evidence that connective tissue disorders (many of which are regarded as autoimmune diseases) occur approximately one year after the onset of prolonged or chronic fatigue, with the implication that fatigue is not merely a symptom of these disorders but precedes them. Many workers have found changes in the immune system of subjects suffering from CFS (mainly immune activation) which could be conducive..." @default.
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- W2015223821 date "1998-01-01" @default.
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- W2015223821 title "Fatigue Syndromes and the Aetiology of Autoimmune Disease" @default.
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- W2015223821 doi "https://doi.org/10.1300/j092v04n04_04" @default.
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