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- W2015387933 abstract "demonstrated that in rats with 5/6 renal ablation block- In response to Dr. Porush’s criticisms of the conclu-ade of the renin-angiotensin system (RAS) by either an sions of our study [1], we submit the following facts forangiotensin-converting enzyme (ACE) inhibitor or by his consideration. Several lines of evidence indicate thatan angiotensin receptor antagonist provides renoprotec- an exaggerated glomerular transmission of hypertensiontion, proportionate to the degree of blood pressure (BP) plays a major role in the pathogenesis of the progressivelowering, which was dose dependent [1]. The authors glomerulosclerosis (GS) observed in the 5/6 ablationinterpret the data to suggest that the renoprotection is model. Moreover, there is an excellent direct correlationonlythroughBP-dependentmechanisms.Infact,thetitle between precisely quantitated BP (continuous radiote-of the article suggests that there was a lack of evidence lemetry) and GS indicating that 60 to 80% of the differ-of BP-independent protection by RAS blockade after ences in GS between individual untreated animals canrenal ablation in this study. They appear to justify these be accounted for by BP differences (" @default.
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- W2015387933 date "2000-10-01" @default.
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- W2015387933 title "Renoprotection and blood pressure" @default.
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- W2015387933 doi "https://doi.org/10.1046/j.1523-1755.2000.00348.x" @default.
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