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- W2015527206 abstract "Pathogenesis of childhood nephrotic syndrome, whether the lesion is minimal change (MCD) or focal segmental glomerulosclerosis (FSGS), remains elusive. Based on the presence of elevated cytokine levels in the peripheral blood, a T-cell induced injury could be postulated. To test the hypothesis that infiltrating T-cell actively contribute to the glomerular injury in children with nephrotic syndrome (NS), we have studied the intrarenal transcription of various T-cell related chemokines, cytokines and cytotoxic T-lymphocyte (CTL) effector molecules in the renal biopsy tissue of 53 children with a variety of histologic lesions. These lesions included MCD (n=7), FSGS (n=20), IgM nephropathy (n=7), lupus nephropathy (n=10), and others (IgA, etc., (n=9)). The biopsies were performed for diagnostic or therapeutic purposes. Two core samples of tissue were obtained. One core was submitted to the pathology lab for morphological diagnosis; the other core was placed in a 1 ml NUNC® cryo tube, sealed, immediately snap frozen in liquid nitrogen and stored at-70° Celsius, then shipped, on dry ice, to the molecular laboratory for gene transcript studies. Intrarenal gene expression was studied using reverse transcription assisted RT PCR." @default.
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- W2015527206 date "1998-04-01" @default.
- W2015527206 modified "2023-10-18" @default.
- W2015527206 title "Evidence of Tissue Growth Factor Beta TGF-β1 - Induced Sclerosis in the Glomeruli of Children with Focal Segmental Glomerulosclerosis (FSGS)• 1843" @default.
- W2015527206 doi "https://doi.org/10.1203/00006450-199804001-01866" @default.
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