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• W2015596811 abstract "The actions of nicotine upon the heart are numerous and varied, affecting rate,¹contractile force,^1-3and, in all likelihood, coronary flow.⁴It is generally considered that a large proportion of this alkaloid's cardiovascular affects are mediated via the autonomic nervous system¹in accordance with nicotine's well-established¹capacity to stimulate, then paralyze all autonomic ganglia. In the case of the heart, then, this drug initially produces cardiac slowing as a consequence of vagal stimulation. Subsequently, with paralysis of the vagal ganglia, there characteristically occurs an acceleration of heart rate, associated with release of cardiac stores of catecholamines. The characteristic enhancement of contractile force following nicotine administration has previously¹been attributed to the above-described release of catecholamines. More recent studies³have demonstrated, however, that higher concentrations of the alkaloid produce a positive inotropic effect upon isolated cat papillary muscle and that the effect is unrelated" @default.
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• W2015596811 date "1969-06-01" @default.
• W2015596811 modified "2023-09-25" @default.
• W2015596811 title "Some Effects of Nicotine on Cardiac Automaticity, Conduction, and Inotropy" @default.
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• W2015596811 doi "https://doi.org/10.1001/archinte.1969.00300160097016" @default.
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