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- W2015610421 abstract "Radiolabeled 15-μm microspheres were used to examine alterations in regional CBF and cerebrovascular resistance in response to changes in arterial PCO 2 . Flow measurements were obtained before and 1–3 and 24 h after 12 min of total cerebral ischemia. Striking sensitivity of blood flow in all areas of the central nervous system was shown to changes in arterial PCO 2 between 24 and 50 mm Hg during the control nonischemic period. Following 12 min of total cerebral ischemia, cerebrovascular resistance increased, producing a decrease in regional blood flow when the important controlling variables for CBF were held constant. One to 3 h after total cerebral ischemia, the effect of variations in arterial PCO 2 on cerebral blood flow was almost completely abolished. Within 24 h after total cerebral ischemia, the sensitivity of CBF to changes in PCO 2 was almost completely restored, whereas the secondary severe neurologic deficit remained. Therapeutic interventions following global cerebral ischemia, designed to ameliorate the “no-reflow” phenomenon and minimize residual ischemic neurologic damage, must take into account this marked early postischemic reduction in sensitivity to normally potent cerebrovasodilatory influences." @default.
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- W2015610421 date "1984-09-01" @default.
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- W2015610421 title "Total Cerebral Ischemia: Effect of Alterations in Arterial PCO2 on Cerebral Microcirculation" @default.
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- W2015610421 doi "https://doi.org/10.1038/jcbfm.1984.51" @default.
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