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- W2015616283 abstract "Abstract Hematopoietic stem cell (HSC) recipients are at risk for severe infections as a result of profound hematopoietic and immune suppression. Whereas myeloid recovery takes 4 to 6 weeks, immunologic reconstitution may take 2 years or more before recovery and, thus, the risk for infection is prolonged. Virtually every common infectious agent can cause morbidity and mortality during the transplant course. Because the predominating immune defects change over time, certain organisms are likely to be encountered at particular post-transplant intervals. Early-on prolonged neutropenia predisposes the recipient to bacterial infections and fungal disease. Latent viruses (eg, cytomegalovirus) become reactivated after neutrophil engraftment and cause severe infections because of cellular and humoral immunodeficiency. Acute and/or chronic graft-versus-host disease (GVHD) can further delay immune reconstitution and, as a consequence, prolong the risk for infection. Cyclosporine, tacrolimus, prednisone, methotrexate, and T-cell depletion of the HSC graft (all measures to prevent GVHD) influence the process of immune recovery. Therefore, to properly evaluate the seriously ill transplant recipient, appreciation of the patient's immune status and its relationship to potential infection is critical. For the HSC transplant patient at high risk for infection, appropriate prevention, aggressive surveillance, early diagnosis, and comprehensive treatment are the most effective measures to reduce morbidity and mortality. Copyright © 2000 by W.B. Saunders Company" @default.
- W2015616283 created "2016-06-24" @default.
- W2015616283 creator A5017848480 @default.
- W2015616283 creator A5076234152 @default.
- W2015616283 date "2000-01-01" @default.
- W2015616283 modified "2023-09-26" @default.
- W2015616283 title "Life-threatening infections in bone marrow transplant patients" @default.
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- W2015616283 doi "https://doi.org/10.1053/spid.0110059" @default.
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