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• W2015751251 abstract "poster) presented at the American Association of Poison Control Centers/American Academy of Clinical Toxicology/ American Board of Medical Toxicology/Canadian Association of Poison Control Centres Annual Scientific Meeting, Sept. 25-30, 1986, Santa Fe, New Mexico. Submitted for publication March 31, 1987; accepted June 10, 1987. Reprint requests: Christopher H. Linden, MD, Assistant Professor, Department of Medicine, University of Massachusetts Medical Center, Worcester, MA 01605. another hospital. Inspection of the vomitus revealed both intact pills and pill fragments. Approximately 11/2 hours after ingestion, vital signs were as follows: pulse 120 beats/min, blood pressure 110/80 mm Hg, and respiratory rate 16/min. The child was flaccid, pale, and responsive only to pain. An intravenous infusion of dextrose 5% in water was started and oxygen was administered by nasal cannula. After gastric lavage with 0.9% saline solution through a size 12F nasogastric tube, the patient was given 5 g of activated charcoal and transferred to our institution. Approximately 3 hours after ingestion, vital signs were as follows: pulse 140 beats/min, blood pressure, t00/80 mm H g , respiratory rate 32/min, and rectal temperature 37 ~ C. CheyneStokes respirations with occasional periods of apnea lasting up to 10 seconds were noted. Rubbing of the sternum abruptly terminated the apnea and resulted in crying and withdrawal of the BUN SGOT SGPT LDH hpf Blood urea nitrogen Serum glutamic-oxaloacetic transaminase Serum glutamic-pyruvic transaminase Lactate dehydrogenase High-power field extremities. Results of the physical examination were otherwise urlchanged from those described above. Gastric lavage was repeated, and 15 g of activated charcoal with 15 ml of 70% sorbitol was administered by nasogastric tube. A Foley catheter was inserted, and the child was admitted to the intensive care unit. Laboratory evaluation yielded the following values: a[terial pH 7.27, Pr 36 mm Hg, and PO~ 110 mm Hg; serum sodium 140 mEq/L, potassium 3.6 mEq/L, chloride 106 mEq/L, bicarbonate 19 mEq/L, anion gap 15 mEq/L, and glucose 219 mg/dl (12.2 mmol/L). The complete blood cell count, the BUN, serum creatinine, SGOT, SGPT, LDH, and alkaline phosphatase levels, the prothrombin and partial thromboplastin times, and the urinalysis results were normal except for glucosuria (3+). Toxicologic analysis of blood and urine by thin-layer chromatography showed positive results only for ibuprofen. No salicylate was detected." @default.
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• W2015751251 date "1987-12-01" @default.
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• W2015751251 title "Metabolic acidosis after acute ibuprofen overdosage" @default.
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• W2015751251 doi "https://doi.org/10.1016/s0022-3476(87)80220-2" @default.
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