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- W2015957793 abstract "HE PATHOGENESIS of liver injury and fibrosis involves complicated interactions among different cell populations in the liver, soluble factors, such as cytokines, and the extracellular matrix. Hepatocytes are injured in a number of pathologic processes (chemical, biological and immunological) and the damage initiates fibrogenesis. Kupffer cells probably are primarily responsible for mediating the second phase of hepatocellular necrosis, induced by reactive oxygen intermediates (1,2), and for activating Ito cells, the major cell type responsible for enhanced extracellular matrix production during the fibrogenic process (3). A number of cytokines and other soluble factors, such as tumour necrosis factor-a (TNF-a), interleukin 2 and 6 (IL-2 and IL-6), and nuclear factor-KB (NF-KB) (4) participate in the damage of hepatocytes and sinusoidal endothelial cells; whereas, transforming growth factor-p (TGF-P) and platelet-derived growth factor (PDGF) have been considered to be fibrogenic cytokines and contribute to the activation of Ito cells (5,6)." @default.
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- W2015957793 title "Modification of liposomes for liver targeting" @default.
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- W2015957793 doi "https://doi.org/10.1016/s0168-8278(96)80274-1" @default.
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