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• W2015984390 abstract "Much evidence strongly suggests a positive role for one or more E2F species in the control of exit from G 0 /G 1 . Results described here provide direct evidence that endogenous E2F-1, as predicted, contributes to progression from G 0 to S. By contrast, cycling cells lacking an intact E2F-1 gene demonstrated normal cell cycle distribution. Therefore, E2F-1 exerts a unique function leading to timely G 0 exit of resting cultured primary cells, while at the same time being unnecessary for normal G 1 to S phase progression of cycling cells." @default.
• W2015984390 created "2016-06-24" @default.
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• W2015984390 date "1998-12-22" @default.
• W2015984390 modified "2023-09-24" @default.
• W2015984390 title "Endogenous E2F-1 promotes timely G ₀ exit of resting mouse embryo fibroblasts" @default.
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• W2015984390 doi "https://doi.org/10.1073/pnas.95.26.15583" @default.
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