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- W2016041305 endingPage "381" @default.
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- W2016041305 abstract "Despite their essential role in host protection, immunoglobulins are also involved in autoimmune processes where antibodies recognize the host's own tissue, triggering inflammatory responses that result in extensive tissue damage. A complex interaction of genetic predisposition, together with environment factors, is thought to trigger immune dysfunction. Although recent studies have dissected the essential role of Fc receptors in autoimmune antibody mediated processes, the uniquely human FcgammaRIIa has not been studied in detail. This Fc receptor is of particular interest, as it is the most abundantly expressed Fc receptor in humans and is implicated in immune complex disease. Investigation of its role has been hampered to date due to lack of suitable animal models. This review examines the evidence for the direct role of this receptor in diseases such as systemic lupus erythematosus and rheumatoid arthritis." @default.
- W2016041305 created "2016-06-24" @default.
- W2016041305 creator A5001063223 @default.
- W2016041305 creator A5057970586 @default.
- W2016041305 creator A5087529375 @default.
- W2016041305 date "2003-10-01" @default.
- W2016041305 modified "2023-09-27" @default.
- W2016041305 title "The role of FcγRIIa as an inflammatory mediator in rheumatoid arthritis and systemic lupus erythematosus" @default.
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- W2016041305 doi "https://doi.org/10.1046/j.1440-1711.2003.01182.x" @default.
- W2016041305 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12969325" @default.
- W2016041305 hasPublicationYear "2003" @default.
- W2016041305 type Work @default.