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• W2016114567 abstract "The present study evaluated the possible role of ionotropic glutamate receptors in mediating the inducible preproenkephalin (PPE) mRNA expression in the rat striatum in response to systemic administration of a psychostimulant amphetamine. A single injection of amphetamine (5 mg/kg, i.p.) increased PPE mRNA levels in the dorsal striatum as revealed via quantitative in situ hybridization. Pretreatment with the NMDA receptor antagonist, MK801 (0.5 mg/kg, i.p.) or 3-((R)-2-carboxypiperazin-4-yl)-propyl-1-phosphonic acid (10 mg/kg, i.p.), attenuated the amphetamine-induced PPE mRNA expression. A similar attenuation of the amphetamine-induced PPE expression, although to a lesser extent, was also observed following pretreatment with a kainate/AMPA receptor antagonist, 6,7-dinitroquinoxaline-2,3-dione (100–200 mg/kg, i.p.). Three antagonists alone had no significant effect on basal levels of PPE mRNAs in the dorsal striatum. These data indicate that both NMDA and kainate/AMPA receptors are involved in coupling extracellular signals evoked by acute stimulant exposure to PPE mRNA expression in striatal neurons." @default.
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• W2016114567 date "2003-07-01" @default.
• W2016114567 modified "2023-10-16" @default.
• W2016114567 title "Contribution of ionotropic glutamate receptors to acute amphetamine-stimulated preproenkephalin mRNA expression in the rat striatum in vivo" @default.
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• W2016114567 doi "https://doi.org/10.1016/s0304-3940(03)00542-1" @default.
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