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• W2016116473 abstract "While sickle cell disease and other chronic hereditary and acquired hemolytic anemias are considered hypercoagulable states, a unifying mechanism explaining the hemostatic activation has been elusive.[1][1] Patients with sickle cell disease exhibit increased thrombin and fibrin generation,[2][2], [3" @default.
• W2016116473 created "2016-06-24" @default.
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• W2016116473 date "2008-01-01" @default.
• W2016116473 modified "2023-10-15" @default.
• W2016116473 title "Hemolysis-associated hypercoagulability in sickle cell disease: the plot (and blood) thickens!" @default.
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• W2016116473 doi "https://doi.org/10.3324/haematol.12318" @default.
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