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• W2016156684 abstract "1. The mechanism of vasodilatation induced by tachykinin peptides was studied in isolated mesenteric arteries of rats. 2. Senktide, a selective NK3 agonist, elicited potent endothelium-dependent relaxation of arteries precontracted with phenylephrine (10(-5) M), but an NK1 agonist did not. 3. A non-peptide NK3 antagonist, SR 142801, inhibited senktide-induced relaxation. However, a non-peptide NK1 antagonist, CP-96,345, and a peptide-based NK2 antagonist, L-659,877, had no effect on senktide-induced relaxation. 4. N omega-nitro-L-arginine (L-NOARG), a nitric oxide synthesis inhibitor, markedly attenuated the relaxant response to senktide. 5. These results suggest that the endothelium of rat mesenteric arteries possesses tachykinin NK3 receptors, and that NK3 agonist-induced vasodilatation is mediated by release of nitric oxide (NO) from the endothelium." @default.
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• W2016156684 date "1995-12-01" @default.
• W2016156684 modified "2023-09-27" @default.
• W2016156684 title "Nitric oxide is a mediator of tachykinin NK3 receptor-induced relaxation in rat mesenteric artery" @default.
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• W2016156684 doi "https://doi.org/10.1111/j.1476-5381.1995.tb15945.x" @default.
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