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- W2016258443 abstract "Prazosin inhibited contractions o isolated-electrically stimulated dog pulmonary artery preincubated with (−)-7-3H-norepinephrine, but had no effect on 3H-overflow, whereas yohimbine enhanced both responses. In pithed dogs the cumulative doses of yohimbine required for 50% inhibition of the pressor effects of clonidine, phenylephrine and norepinephrine were 23, 188 and 35 μg/kg i.v., and those for prazosin were 9, 15 and 5 μg/kg i.v., respectively. The agonists could be ranked in pairs depending on the extent to which the antagonists discriminated membranes of the pair: clonidine-phenylephrine > phenylephrine-norepinephrine>clonidine-norepinephrine. The results suggest that pressor effects in dogs are mediated by both α1- and α2-vascular (i.e. postsynaptic) adrenoceptors." @default.
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- W2016258443 title "α2- and α2-vasular adrenoceptors in the dog" @default.
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- W2016258443 doi "https://doi.org/10.1016/0014-2999(80)90460-4" @default.
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