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• W2016283456 endingPage "210" @default.
• W2016283456 startingPage "194" @default.
• W2016283456 abstract "Severe cutaneous adverse reactions (SCARs), such as Stevens-Johnson syndrome (SJS) and toxic epidermal necrosis (TEN), are rare but life-threatening conditions induced mainly by a variety of drugs. Until now, an effective treatment for SJS/TEN still remains unavailable. Current studies have suggested that the pathobiology of drug-mediated SJS and TEN involves major histocompatibility class (MHC) I-restricted activation of cytotoxic T lymphocytes (CTLs) response. This CTLs response requires several cytotoxic signals or mediators, including granulysin, perforin/granzyme B, and Fas/Fas ligand, to trigger extensive keratinocyte death. In this article, we will discuss the cytotoxic mechanisms of severe cutaneous adverse reactions and their potential applications on therapeutics for this disease." @default.
• W2016283456 created "2016-06-24" @default.
• W2016283456 creator A5039567335 @default.
• W2016283456 creator A5085542552 @default.
• W2016283456 date "2014-01-03" @default.
• W2016283456 modified "2023-10-14" @default.
• W2016283456 title "Cytotoxic Proteins and Therapeutic Targets in Severe Cutaneous Adverse Reactions" @default.
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