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- W2016299182 abstract "Introduction Septic shock results when infectious agents or infectioninduced mediators in the bloodstream produce hemodynamic decompensation. Standard definitions of graded response to infection now exist (Table 1). Septic shock is primarily a form of distributive shock and is characterized by ineffective tissue oxygen delivery and extraction associated with inappropriate peripheral vasodilatation despite preserved or increased cardiac output. In septic shock, a complex interaction between pathologic vasodilatation, relative and absolute hypovolemia, myocardial dysfunction, and altered blood flow distribution occurs because of the inflammatory response to infection. Even after restoration of intravascular volume, microcirculatory abnormalities may persist. Half of the patients who die with septic shock experience multiple organ system failure. Most of the rest have progressive hypotension with low systemic vascular resistance, which is unresponsive to vasopressor agents. Myocardial dysfunction is not uncommon; however, death of myocardial failure is rare.1-3" @default.
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- W2016299182 date "2007-09-01" @default.
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- W2016299182 title "Cardiovascular support in septic shock" @default.
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- W2016299182 doi "https://doi.org/10.1016/j.amj.2007.06.001" @default.
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