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• W2016395855 abstract "The mechanisms underlying cell damage in stroke or during experimental brain ischemia are not fully understood.l-Cyteine, an excitotoxic amino acid that could contribute to tissue damage, is normally found in relatively low levels in brain (ca. 0.05 μmol/g), compared to the cysteine-containing tripeptide, glutathione (GSH, ca. 1.5 μmol/g). We have observed that during brain ischemia in gerbils, levels of cysteine rise 10–13-fold over an 8 h period to 0.66 and 0.62 μmol/g, respectively, in the ischemic hippocampus and striatum. At the same time, levels of GSH fall by 0.84 and 0.94 μmol/g, respectively. The elevated free cysteine may be derived largely from GSH. The levels of cysteine found in ischemic brain are similar to those reported after parenteral administration of neurtoxic doses ofl-cysteine to perinatal rats. The remarkable increase in cysteine during brain ischemia, coupled to its neurotoxic properties, may play a role in aspects of brain damage during or following brain ischemia." @default.
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• W2016395855 date "1993-04-01" @default.
• W2016395855 modified "2023-10-16" @default.
• W2016395855 title "Brain ischemia markedly elevates levels of the neurotoxic amino acid, cysteine" @default.
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• W2016395855 doi "https://doi.org/10.1016/0006-8993(93)90770-n" @default.
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