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- W2016461063 endingPage "11" @default.
- W2016461063 startingPage "1" @default.
- W2016461063 abstract "Alzheimer's disease (AD) is the most frequent neurodegenerative disorder leading to dementia in the aged human population. It is characterized by the presence of two main pathological hallmarks in the brain: senile plaques containing beta-amyloid peptide and neurofibrillary tangles (NFTs), consisting of fibrillar polymers of abnormally phosphorylated tau protein. Both of these histological characteristics of the disease have been simulated in genetically modified animals, which today include numerous mouse, fish, worm, and fly models of AD. The objective of this review is to present some of the main animal models that exist for reproducing symptoms of the disorder and their advantages and shortcomings as suitable models of the pathological processes. Moreover, we will discuss the results and conclusions which have been drawn from the use of these models so far and their contribution to the development of therapeutic applications for AD." @default.
- W2016461063 created "2016-06-24" @default.
- W2016461063 creator A5047620994 @default.
- W2016461063 creator A5069146400 @default.
- W2016461063 creator A5079838423 @default.
- W2016461063 creator A5084018006 @default.
- W2016461063 date "2010-01-01" @default.
- W2016461063 modified "2023-10-17" @default.
- W2016461063 title "Neuronal Models for Studying Tau Pathology" @default.
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- W2016461063 doi "https://doi.org/10.4061/2010/528474" @default.