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• W2016566711 abstract "Background and purpose There is limited knowledge regarding the long‐term outcome of the methcathinone/manganese‐induced movement disorder. Our purpose was to define prognosis in intravenous methcathinone abusers affected by this distinctive disorder attributed to manganese ( M n) toxicity. Also, neuropathology from a globus pallidus region biopsy from a former user is reported. Methods Eighteen methcathinone abusers were categorized as active (five), discontinued (four) or former (nine) users. They were reassessed after a median of 32.5 months (range 3.4–59.6) clinically, on rating scales, and with MRI and blood M n levels. The biopsy was examined ultrastructurally. Results Overall the group showed a slight tendency to deterioration at follow‐up on clinical assessment of motor functioning, especially the active users. No significant change occurred on parkinsonian rating scale reassessment. Significant reduction in M n levels occurred in former users, and decreased T 1‐weighted hyperintensity on basal ganglia MRI occurred in 3 of 4 former and 2 of 3 discontinued users, despite lack of clinical improvement. The biopsy consisted of white matter showing decompacted myelin sheaths and frequent abnormalities of mitochondria. Conclusions No improvement in this M n‐induced movement disorder occurs after cessation of methcathinone abuse despite improvement of M n blood levels and/or MRI abnormalities. Ultrastructural abnormalities in a former user confirm structural damage to white matter is associated with the disorder. Methcathinone/ M n toxicity is an important, disabling and permanent medical sequel of intravenous drug abuse in the former Soviet Union." @default.
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• W2016566711 date "2013-05-17" @default.
• W2016566711 modified "2023-10-04" @default.
• W2016566711 title "The outcome of the movement disorder in methcathinone abusers: clinical, MRI and manganesemia changes, and neuropathology" @default.
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• W2016566711 doi "https://doi.org/10.1111/ene.12185" @default.
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