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- W2016675620 endingPage "186" @default.
- W2016675620 startingPage "170" @default.
- W2016675620 abstract "Skeletal muscle is a tissue of high demand and it accounts for most of daily energy consumption. The classical concept of energy metabolism in skeletal muscle has been profoundly modified on the basis of studies showing the influence of additional factors (i.e., uncoupling proteins (UCPs) and peroxisome proliferator activated receptors (PPARs)) controlling parameters, such as substrate availability, cellular enzymes, carrier proteins, and proton leak, able to affect glycolysis, nutrient oxidation, and protein degradation. This extremely balanced system is greatly altered by cancer disease that can induce muscle cachexia with significant deleterious consequences and results in muscle wasting and weakness, delaying or preventing ambulation, and rehabilitation in catabolic patients." @default.
- W2016675620 created "2016-06-24" @default.
- W2016675620 creator A5012956702 @default.
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- W2016675620 creator A5064533816 @default.
- W2016675620 creator A5073878691 @default.
- W2016675620 date "2003-08-18" @default.
- W2016675620 modified "2023-10-15" @default.
- W2016675620 title "Skeletal muscle metabolism in physiology and in cancer disease" @default.
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