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- W2016729849 abstract "Accumulating evidence indicates that interferon-β (IFN-β) can modify the complex immunopathogenic scenario causing clinical relapse activity and disease progression in MS. However, the beneficial effects of IFN-β in MS patients may also depend on non-immune mechanisms, including the modulation of astrocyte function. In the present report, we have shown that, depending on the dose, IFN-β treatment can either promote astrocyte proliferation and survival, or result astrocyte death. These actions depend, at least in part, on the regulation of nuclear factor-kappa B (NF-κB), an inducible transcription factor present in neurons and glia. This bimodal effect of IFN-β adds a new layer of complexity in the actions of IFN-β within the CNS." @default.
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- W2016729849 date "2010-09-14" @default.
- W2016729849 modified "2023-09-30" @default.
- W2016729849 title "Bimodal effect of interferon-β on astrocyte proliferation and survival: Importance of nuclear factor-κB" @default.
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- W2016729849 doi "https://doi.org/10.1016/j.jneuroim.2010.05.036" @default.
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