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- W2016992253 abstract "Human red cells deficient in glycophorin B are partly resistant to invasion by Plasmodium falciparum and become completely resistant when glycophorin A is removed from their surface by trypsin treatment. Similar treatment of cells which have a hybrid glycophorin molecule renders them glycophorin-deficient and resistant to invasion. Tn and Wrb - ve cells with defined alterations in glycophorin A or B are also resistant to invasion. These findings suggest that both glycophorins A and B are involved in parasite invasion, indicate which parts of these molecules may be involved in this process, and provide the basis for a tentative model of parasite/red-cell interactions." @default.
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- W2016992253 date "1982-10-01" @default.
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- W2016992253 title "GLYCOPHORIN AS A POSSIBLE RECEPTOR FOR PLASMODIUM FALCIPARUM" @default.
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- W2016992253 doi "https://doi.org/10.1016/s0140-6736(82)90157-x" @default.
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