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- W2017011952 abstract "Previous studies have shown that lateral hypothalamic (LH) lesions produce numerous disorders including aphagia, gastric pathology, hyperthermia, and sensorimotor dysfunctions. Because insulin pretreatments shorten the period of aphagia following LH lesions, the present study examined the possibility that insulin might also alter the incidence of other disorders resulting from LH damage. It was found that insulin pretreatments shortened the period of postoperative aphagia and reduced, but did not prevent, the occurrence of glandular gastric pathology. In addition, insulin did not prevent the hyperthermia or attenuate the severity of sensorimotor dysfunctions. Groups of LH damaged rats pretreated with either insulin or saline and allowed to recover initial feeding behaviors displayed significant reductions in core temperature and less gastric pathology than a group of LH rats also pretreated with saline but which was sacrificed while still aphagic. Furthermore, LH animals which ate did so in the presence of rather pronounced sensorimotor abnormalities. These findings suggest that aphagia and sensorimotor dysfunctions induced by LH lesions can be dissociated. They further suggest that viscero-metabolic disroders may play an important role in the initial stages of the LH syndrome, and indicate that insulin may influence recovery of feeding behaviors by affecting peripheral systems as well as central nervous system functions." @default.
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- W2017011952 date "1980-12-01" @default.
- W2017011952 modified "2023-09-27" @default.
- W2017011952 title "Aphagia, gastric pathology, hyperthermia, and sensorimotor dysfunctions following lateral hypothalamic lesions: Effects of insulin pretreatments" @default.
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- W2017011952 doi "https://doi.org/10.1016/0031-9384(80)90313-3" @default.
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