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- W2017152304 abstract "Altered expression or activity of histone lysine methylases and demethylases in cancer lead to aberrant chromatin modification patterns, which contribute to uncontrolled cell proliferation via cancer-specific deregulation of gene expression programs or the induction of genome instability. Several transcription factors that regulate growth-associated genes undergo lysine methylation, expanding the repertoire of regulatory targets modulated by histone-methylating enzymes during tumorigenesis. In certain specific tumor types or specific physiological conditions, these enzymes may trigger chromatin structure and/or transcription factor activity changes that result in opposite effects on cancer initiation or progression. The mechanisms of such context-specific dual functions and those involved in the crosstalk between factor and histone modifications are subject to extensive research, which is beginning to shed light into this novel level of complexity of cancer-related epigenetic pathways." @default.
- W2017152304 created "2016-06-24" @default.
- W2017152304 creator A5037092248 @default.
- W2017152304 creator A5046268636 @default.
- W2017152304 creator A5054635696 @default.
- W2017152304 date "2013-03-18" @default.
- W2017152304 modified "2023-09-26" @default.
- W2017152304 title "Context-specific regulation of cancer epigenomes by histone and transcription factor methylation" @default.
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