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• W2017156351 endingPage "344" @default.
• W2017156351 startingPage "336" @default.
• W2017156351 abstract "When the benzodiazepine inverse agonist DMCM (6,7-dimethoxy-4-ethyl-beta-carboline-3-carboxylic acid methyl ester) occupies the benzodiazepine recognition site on the GABAA receptor complex, the inhibitory action of gamma-aminobutyric acid (GABA) is attenuated. DMCM acted as an unconditional stimulus for one response associated with fear or anxiety, analgesia, as indicated by a dose-dependent (0.25-1.0 mg/kg) suppression of rats' responses to a formalin injection. This was accompanied by other fearlike responses (defecation and urination). The opioid antagonist naltrexone (1.75-14 mg/kg) did not affect these behaviors. Environmental cues associated with DMCM provoked analgesia and defecation in the absence of the drug. The conditional analgesia was reversed by naltrexone (7 mg/kg). DMCM functions as an unconditional fear stimulus by eliciting fear-related behaviors and conditioning those responses to neutral stimuli. The neural circuitry underlying fear conditioning appears to involve tonically inhibitory GABAergic synapses." @default.
• W2017156351 created "2016-06-24" @default.
• W2017156351 creator A5054185030 @default.
• W2017156351 creator A5058421568 @default.
• W2017156351 date "1992-01-01" @default.
• W2017156351 modified "2023-10-14" @default.
• W2017156351 title "The benzodiazepine inverse agonist DMCM as an unconditional stimulus for fear-induced analgesia: Implications for the role of GABA-sub(A ) receptors in fear-related behavior." @default.
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• W2017156351 doi "https://doi.org/10.1037//0735-7044.106.2.336" @default.
• W2017156351 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1317186" @default.
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