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- W2017264267 abstract "Summary Shortly after myocardial infarction, various circulating leukocyte subsets accumulate in the heart. Leukocyte recruitment is highly coordinated and relies on cell production in the bone marrow, mobilization to the blood, and chemokine-mediated infiltration to the destination tissue. Neutrophils, which are phagocytic and inflammatory, are among the first leukocytes to accumulate in large numbers. Within a day, neutrophils disappear and are replaced by a subset of monocytes that further contribute to inflammation and phagocytosis. After a few days, monocyte-derived reparative macrophages accrue, quell inflammation, and foster angiogenesis and tissue remodelling. Studies suggest a wellbalanced response comprising these three waves is essential to optimal infarct healing." @default.
- W2017264267 created "2016-06-24" @default.
- W2017264267 creator A5034311334 @default.
- W2017264267 date "2015-01-01" @default.
- W2017264267 modified "2023-09-23" @default.
- W2017264267 title "Inflammation and repair in the ischaemic myocardium" @default.
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- W2017264267 doi "https://doi.org/10.5482/hamo-14-09-0045" @default.
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