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- W2017287483 abstract "The ability of ß-endorphin (ß-END) to release prolactin and the ability of naloxone to block prolactin's release when delivered to specific hypothalamic areas via push-pull perfusion was studied in unrestrained, conscious, Ovariectomized rats. Perfusion of either the arcuate nucleus or the preoptic area with ß-END for 15 to 30 min caused a large, brief increase in plasma prolactin levels. Perfusion for a longer time period (120 min) resulted in peak prolactin levels at 60 min, with a return to baseline by 120 min, suggesting that ß-END primarily acts to induce a sequence of events that culminates in prolactin release, but other factors are needed to maintain this release over long periods of time. Perfusion of the arcuate nucleus for two 15-min periods 90 min apart resulted in two surges of prolactin. When naloxone, the opiate receptor antagonist, was added to the perfusate, ß-END was not capable of stimulating prolactin release. These results provide a model to answer whether endogenous ß-END has a role in the neuroendocrine regulation of prolactin surges and what the location is of the opiate neurons involved in this neuronal pathway." @default.
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- W2017287483 title "Arcuate Nucleus and Preoptic Area Involvement in Beta-Endorphin-lnduced Release of Prolactin in Conscious Ovariectomized Rats" @default.
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- W2017287483 doi "https://doi.org/10.1111/j.1365-2826.1989.tb00146.x" @default.
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