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- W2017335922 abstract "H2O2 (20–200 μM) elicited concentration-dependent contraction of rat isolated aortic rings. Pretreatment of the preparations with N-nitro-L-arginine (L-NNA) or removal of the endothelium augmented significantly the contractile response of rings to H2O2. The increase of the extracellular potassium concentration ([K+]o) resulted in a K+-dependent increase of the contractile response. In Ca2+-free physiological salt solution (PSS), responses to H2O2 were decreased approximately to 4% of the 100 mM KCl-induced response. In addition, calcium channel blockers (nisoldipine, nifedipine, verapamil, flunarizine) did not completely inhibit H2O2-induced contractions. H2O2-evoked contractions were abolished after repeated applications of caffeine or noradrenaline in Ca2+-free PSS. Both extracellular Ca2+entry through voltage-dependent Ca2+channels and intracellular Ca2+ from caffeine- and noradrenaline-sensitive stores appear to participate in H2O2-induced contractions of rat aortic rings." @default.
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- W2017335922 date "1998-07-01" @default.
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- W2017335922 title "Investigation of the Mechanisms Underlying H2O2-Evoked Contraction in the Isolated Rat Aorta" @default.
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- W2017335922 doi "https://doi.org/10.1016/s0306-3623(97)00392-3" @default.
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