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- W2017372544 abstract "Our view of hair follicle morphogenesis and cycling is that it is orchestrated by changes in the status of the activity of TCF/LEF1-regulated genes, which are at least in part regulated by Wnt signaling. In turn, a number of other key signaling pathways, including those triggered by BMPs, FGFs, and Shh, participate in activating downstream nuclear regulatory factors, such as Hoxc13, Msx1, Msx2, Whn, hairless, RXRα, SMADs, and Soxes. Why would nature select Wnts to conduct this symphony? While the answer remains unknown, β-catenin has a Jeckyl and Hyde life—sometimes it functions in cell-cell adhesion, and other times it masquerades as a transcription factor. On the surface, it seems simpler to design a brand new gene encoding an entire transcription factor rather than to reuse β-catenin to provide a transactivation domain for LEF1." @default.
- W2017372544 created "2016-06-24" @default.
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- W2017372544 date "2001-07-01" @default.
- W2017372544 modified "2023-10-16" @default.
- W2017372544 title "At the Roots of a Never-Ending Cycle" @default.
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- W2017372544 doi "https://doi.org/10.1016/s1534-5807(01)00022-3" @default.
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