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- W2017411471 abstract "Hemorrhagic shock (HS) in trauma patients results in liver injury from impaired blood flow and increased mediators of the systemic inflammatory response. These mediators include high-mobility group protein B1 (HMGB1), a marker of hepatocyte necrosis, and toll-like receptor (TLR)-4 and TLR-9, which are thought to initiate early and late inflammation in the liver during resuscitated HS. A new treatment strategy, direct peritoneal resuscitation (DPR), improved liver blood flow, function, and survival after HS." @default.
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- W2017411471 date "2013-02-01" @default.
- W2017411471 modified "2023-09-27" @default.
- W2017411471 title "Direct Peritoneal Resuscitation Prevents Cytoplasmic HMGB1 after Hemorrhagic Shock" @default.
- W2017411471 doi "https://doi.org/10.1016/j.jss.2012.10.645" @default.
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