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- W2017442957 abstract "THE central role of iodine deficiency in the etiology of simple goiter has been firmly established (Marine, 1935). Thyroid hyperplasia readily develops in rats maintained on a goitrogen-free low iodine diet (Levine et al, 1933, Remington and Remington, 1938). According to the most generally accepted theory of the pathogenesis of thyroid changes in iodine deficiency, the inadequate supply of iodine curtails the formation of thyroid hormone, and the resulting hypothyroid state leads to compensatory hypersecretion of pituitary thyrotrophin (TSH) (Marine, 1935; Greer, 1950; Riggs, 1952). In addition to thyroid enlargement, further signs of increased TSH production were described in rats fed low iodine diets, such as augmented thyroid cell height, pituitary changes similar to those seen in hypothyroid animals and enhanced thyroidal radioiodine collection (Leblond and Mann, 1942). Chapman (1941), however, reported that thyroid cell height increases in response to low iodine intake even in the absence of the pituitaiy." @default.
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- W2017442957 date "1954-02-01" @default.
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- W2017442957 title "REGULATION OF THE RAT THYROID IN SHORTTERM IODINE DEFICIENCY1" @default.
- W2017442957 doi "https://doi.org/10.1210/endo-54-2-216" @default.
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