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- W2017831286 abstract "Histamine activation of H1 receptors elicited the release of adrenaline from in vitro perfused rat adrenal with an EC50 of 3 microM. Neither Na+ deprivation nor complete membrane depolarization abolished the histamine-mediated secretory response but it was partially dependent on extracellular Ca2+. Nitrendipine and BAY-K-8644 affected the release induced by histamine concentrations at over 3 microM. Delayed application of histamine pulses, after external Ca2+ removal, led to a decline in to a plateau at 50% of the initial release. Pretreatment with ionomycin abolished this Ca2+ deprivation-resistant component. These data suggest that secretion evoked by low concentrations of histamine occurs by mobilization of Ca2+ from internal stores whereas higher concentrations use Ca2+ from both intracellular and extracellular sources." @default.
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- W2017831286 date "1993-09-01" @default.
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- W2017831286 title "Ionic mechanisms involved in the secretory effects of histamine in the rat adrenal medulla" @default.
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- W2017831286 doi "https://doi.org/10.1016/0014-2999(93)90202-s" @default.
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