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- W2018011149 abstract "Chemokines and their receptors have been studied in several solid tumor models as mediators of inflammation. In turn, inflammation has been implicated in the promotion and progression of tumors, and as such, chemokines have been proposed as novel molecular targets for chemotherapy. While the expression of these molecules has been described in tumor cells, endothelial cells, macrophages and neutrophils, less attention has been paid to the expression profile of these molecules by T lymphocytes in the periphery or infiltrating the tumor. Using the D1-DMBA-3 murine mammary adenocarcinoma model, we aimed to better characterize the differential expression of chemokines and/or their receptors in the host and in the tumor microenvironment, and specifically, in the T cells of tumor-bearing mice compared to normal control animals. We found that T lymphocytes from tumor-bearing mice express the pro-inflammatory chemokines, CCL2, CCL5 and CXCL2, as well as the chemokine receptors, CCR1, CCR2, CCR3 and CXCR2." @default.
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- W2018011149 date "2009-04-01" @default.
- W2018011149 modified "2023-10-14" @default.
- W2018011149 title "506 THE RAS INHIBITOR FARNESYLTHIOSALICYLIC ACID INHIBITS THE GROWTH OF HEPATOCARCINOMA CELL LINES IN VITRO AND IN VIVO" @default.
- W2018011149 doi "https://doi.org/10.1016/s0168-8278(09)60508-0" @default.
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