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- W2018214174 endingPage "164" @default.
- W2018214174 startingPage "156" @default.
- W2018214174 abstract "Mutations in the autoimmune regulator (AIRE) protein are the causative factor in development of the human disease autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy (APECED). In mice, the absence of the analogous protein aire influences ectopic expression of peripheral tissue antigens in thymic medullary epithelial cells (MECs), resulting in the development of an autoimmune disorder similar to APECED and establishing aire/AIRE as an important player in the induction of central tolerance. However, the molecular mechanism of AIRE's function, in particular its ability to specifically control the expression of peripheral tissue antigens in MECs, is still unclear. Here, we review current evidence relating to the molecular mechanism of AIRE." @default.
- W2018214174 created "2016-06-24" @default.
- W2018214174 creator A5040731525 @default.
- W2018214174 creator A5070997333 @default.
- W2018214174 creator A5078237329 @default.
- W2018214174 date "2005-03-16" @default.
- W2018214174 modified "2023-10-15" @default.
- W2018214174 title "AIRE and APECED: molecular insights into an autoimmune disease" @default.
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