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- W2018267556 abstract "The investigation of spontaneous as well as induced asthma has often centered on studies of the pathophysiology of airflow obstruction.‘, 2 An increase in airflow resistance alone does not explain all of the clinical and physiologic changes characteristic of an asthmatic attack such as: a decrease in vital capacity, hyperinflation, transient loss of lung elastic recoil, or severe dyspnea.“-’ During spontaneous exacerbations of asthma, asthmatics not only increase functional residual capacity but many patients also develop large increases in their total lung capacity.’ In 1 patient reported by Woolcock and Read,” the residual volume during the acute attack was greater than the total lung capacity after recovery. Associated with this hyperinflation are decreases in lung elastic recoil5 which are reversed with treatment and are not as severe as those found in emphysema. Reports in the literature vary about whether similar hyperinflation occurs during the production of acute bronchoconstriction in the experimental laboratory. Some investigators have not shown increases in total lung capacity with either exercise-induced asthma83 9 or by bronchoconstrictor aerosols such as antigenlO I1 or histamine.” Others have shown that significant hyperinflation and loss of lung elastic recoil can be produced by inhaling bronchoconstrictor agents12, I3 and during postexertional asthma. I4" @default.
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- W2018267556 title "Physiologic effects of inhaled histamine in asthma: Reversible changes in pulmonary mechanics and total lung capacity" @default.
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- W2018267556 doi "https://doi.org/10.1016/0091-6749(79)90020-4" @default.
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