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- W2018634835 abstract "Transformation of human cells requires both SV40 large T and small t antigens. Plasmids that contained mutations in the amino-terminal dnaJ domain of the early region fail to transform human diploid fibroblasts. However, large T dnaJ mutants can be rescued by plasmids that express early region products other than large T antigen. The protein found to be responsible for such complementation was the third early region product, 17KT. Similar to large T, this protein reduces levels of the retinoblastoma-related protein, p130, and stimulates cell-cycle progression of quiescent fibroblasts, two activities of large T that are disrupted by dnaJ mutations." @default.
- W2018634835 created "2016-06-24" @default.
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- W2018634835 date "2003-10-01" @default.
- W2018634835 modified "2023-10-18" @default.
- W2018634835 title "SV40 17KT antigen complements dnaj mutations in large T antigen to restore transformation of primary human fibroblasts" @default.
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- W2018634835 doi "https://doi.org/10.1016/s0042-6822(03)00524-5" @default.
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