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- W2018705611 abstract "Abstract Chronic and acute elevations in pulmonary vascular resistance remain a major source of morbidity and mortality following surgical correction of congenital heart disease [1]. Chronic elevation in pulmonary vascular resistance results from structural changes of the pulmonary vascular bed, which decreases cross-sectional area. After successful surgery, if these structural changes are reversible, pulmonary vascular resistance will begin to decrease in the postoperative period. Acute elevation in pulmonary vascular resistance results from the active contraction of structurally abnormal pulmonary arteries during a period of extreme vasoreactivity in the immediate postoperative period [2]. This decreases cardiac output, produces acidosis and hypoxemia and may result in significant morbidity and mortality. This period of extreme vasoreactivity is caused by the degranulation of platelets and leukocytes following cardiopulmonary bypass and hypothermia, with the subsequent release of potent vasoconstrictors [3]. The postoperative management of patients with preoperative increases in pulmonary arterial pressure and pulmonary vascular resistance is directed at preventing pulmonary hypertensive crises during this period of extreme vasoreactivity." @default.
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- W2018705611 date "1995-08-01" @default.
- W2018705611 modified "2023-09-27" @default.
- W2018705611 title "Pulmonary vascular regulation in newborns, infants and children after surgery for congenital heart disease" @default.
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- W2018705611 doi "https://doi.org/10.1016/1058-9813(95)00121-i" @default.
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