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- W2018723169 abstract "There is increasing evidence that prostaglandins are mediators of inflammation in skin and that prostaglandins are synthesised locally in response to the inflammatory stimulus. The effect of four nonsteroid anti-inflammatory or antipyretic drugs on prostaglandin biosynthesis by human skin has therefore been studied. Aspirin (0.56 mM) and indomethacin (0.28 mM) produced a small but significant inhibition of synthesis of prostaglandin E2. Indomethacin and chloroquine, but not aspirin, inhibited synthesis of prostaglandin F2a. Acetaminophen inhibited synthesis of prostaglandin F2, but did not inhibit prostaglandin E2 synthesis. None of the drugs studied are therapeutically effective anti-inflammatory agents in human skin and it may be significant that the inhibitory effects of aspirin and indomethacin on prostaglandin synthesis by skin are small compared with the effects of the same drugs on prostaglandin synthesis in other tissues." @default.
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- W2018723169 title "Effect of Nonsteroid Anti-Inflammatory and Antipyretic Drugs on Prostaglandin Biosynthesis by Human Skin" @default.
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- W2018723169 doi "https://doi.org/10.1111/1523-1747.ep12676186" @default.
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