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- W2018847596 abstract "Bay K8644 increased unidirectional Ca2+ influx and produced tension development in rabbit aorta. Both responses could be evoked in the tissue maximally stimulated with norepinephrine. When the arterial rings were maximally activated by high K+ depolarization, Bay K8644 was without effect. The tension evoked by high K+ and Bay K8644 was more sensitive to the dihydropyridine Ca2+ antagonist PY108-068 than norepinephrine induced tension. These results indicate that Bay K8644 activates only potential operated Ca2+ channels which are opened by high K+ depolarization." @default.
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- W2018847596 title "Bay K8644 differentiates between potential and receptor operated Ca2+ channels" @default.
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- W2018847596 doi "https://doi.org/10.1016/0014-2999(84)90581-8" @default.
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